An EM Approach to Syncope in Adults
Medical Student Pearl
Med 3
Memorial University of Newfoundland Class of 2024
Reviewed by Dr. J Gross
Copy Edited by Dr. J Vonkeman
Pdf Download: EMSJ An EM Approach to Syncope by SFargeria
Case
A 60-year-old male presented to the ED after experiencing recurrent episodes of syncope. The first episode occurred at a convenience store in an upright position. He denied prodrome and exertional activity at the time of syncope. After a transient loss of consciousness, he woke up confused with urinary incontinence. He felt nauseous and had emesis in the ambulance on the way to ED. He had two more episodes of syncope over the span of two hours. On assessment in the ED, he endorsed a past history of light-headedness preceded by laughing and holding his breath. He denied dyspnea and chest pain. He had no significant past medical history. There was no family history of cardiovascular disease and syncope, and social history was unremarkable.
On examination, he was alert and oriented. He had a minor laceration on his forehead from the fall. His respiratory and cardiovascular exams were unremarkable, neurological exam was normal. In the ED, his blood work was unremarkable. He was placed on telemetry when he had two more episodes of syncope. The monitor showed 20-second-long sinus pauses corresponding with the syncopal episodes. Cardiology was consulted and he was temporarily placed on intermittent transcutaneous pacing.
Differential Diagnosis of Syncope2
True Syncope
1. Reflex (autonomic hypersensitivity)
- Vasovagal, carotid sinus hypersensitivity, situational
2. Orthostatic hypotension
- Volume depletion, autonomic failure
3. Cardiac
- Valvular (aortic stenosis, mitral stenosis), dysrhythmias (bradyarrhythmia, ventricular tachyarrhythmia, supraventricular tachyarrhythmia), mechanical (pacemaker dysfunction), cardiomyopathy, infiltrative (eg. hemochromatosis, sarcoidosis, amyloidosis), acute MI, ARVC, cardiac tamponade, acute aortic dissection
Other Causes
1. Medication/ Drug-induced
- Anti-hypertensives, QT prolonging meds, insulin, alcohol, anti-depressants, anti-glycemic agents, diuretics, anti-anginal agents, etc
2. Transient Loss of Consciousness (TLOC)
-
- Traumatic brain injury, seizure disorders, intoxications, hindbrain TIA, conversion disorders and metabolic abnormalities
Background
Syncope is defined as a brief, sudden, transient loss of consciousness due to cerebral hypoperfusion1. The three broad categories of syncope are reflex, orthostatic and cardiac syncope. The most common cause of cardiac syncope includes dysrhythmias1. A good past medical history of cardiovascular disease is important as it is 85-94% sensitive and 64-83% specific in predicting a cardiac etiology of syncope1.
Diagnostic Workup
Diagnostic workup for syncope requires a thorough history, physical exam, and a 12-lead ECG. Cardiac monitoring is necessary in patients that present to ER with an acute presentation of syncope, and a strong suspicion for cardiac etiology2. History should consist of identifying high-risk features that warrant a prompt cardiology consult2. A detailed HPI should consist of asking about an absence of a prodrome, exertional or supine syncope, concomitant trauma, past medical history of cardiovascular disease and family history of sudden cardiac death (<50 years)2. Low-risk features include presence of a prodrome, specific triggers (eg. dehydration, stress, laughter), syncope while upright and the absence of cardiovascular disease2. Vital signs and a cardiac exam should be completed2. If cardiac causes of syncope cannot be ruled out on first assessment, a 12-lead ECG should be placed to assess for dysrhythmias or conduction disease, and serial troponin values should be collected2.
Though there are multiple clinical decision rules for syncope, the following have been externally validated: Evaluation of Guidelines in Syncope Study (EGSYS), San Francisco Syncope Rule and Osservatorio Epidemiologico sulla Sincope nel Lazio (OESIL)1. Patients that are stratified as high risk require admission for further evaluation. EGSYS predicts the probability of cardiac syncope at two years based on abnormal ECG findings (eg. BBB, sinus bradycardia), heart disease (eg. ischemic, structural), palpitations before syncope, as well exertional and positional syncope, symptoms of prodrome (nausea/vomiting) and predisposing/precipitating factors1. An admission is warranted if the patient scores a three or higher as there is a 21% mortality risk at two years1. The OESIL risk score estimates a 1-year all-cause mortality in patients presenting with syncope1. The factors include age (>65), history of cardiovascular disease, lack of prodrome and abnormal ECG characteristics (eg. BBB, AV conduction disorders and hypertrophy)1. Admission is warranted for one or more variables1. The Canadian Syncope Risk Score can be used in patients presenting to ER with syncope to predict a 30-day serious adverse events2. It consists of factors such as abnormal QRS axis, corrected QT interval >480 ms, elevated troponin (>99th percentile of normal population) and ED diagnosis based on evaluation to stratify patients into risk categories: very low (-3 to -2), low (-1 to 0), medium (1 to 3), high (4 to 5) and very high (6 to 11)2.
The Canadian Journal of Cardiology recommends a disposition algorithm for patients presenting to ER with syncope that is based on history of a serious medical condition and high-risk features3. Figure 1 illustrates an approach to disposition from the ER. Patients that have an unclear etiology and intermediate risk should be considered for an urgent cardiology assessment.
Figure 1: A disposition plan for patients presenting to the ER with syncope (Canadian Cardiovascular Society 2020).
Best Practice for Treatment
Given the benign course, treatment for vasovagal syncope is based on lifestyle modification, education and reassurance2. Lifestyle modification consists of educating patients on identifying and managing prodromes early and managing triggers (eg. dehydration, defecation, micturition, laughing, coughing and crowded environments)2.
Treatment for orthostatic syncope also relies on lifestyle modification, education and reassurance2. Lifestyle modification consists of re-adjusting diuretics, ACE-inhibitors, angiotensin receptor blockers, calcium channel and beta blockers to ensure optimal blood pressure and hydration control2.
Managing cardiac syncope requires addressing the underlying etiology through antiarrhythmic medications (eg. tachyarrhythmias), cardiac pacing (eg. bradyarrhythmias), catheter-directed ablation and ICD insertion1. Cardiac pacemaker therapy is indicated for patients that have intermittent sinus node disease if correlation is identified between sinus pauses on ECG and syncope3. Selected patients that are diagnosed with the bradycardia-tachycardia form of sick sinus syndrome, can benefit from a percutaneous cardiac ablative technique3. Dual-chamber pacing is recommended for patients with sinus node dysfunction provided there is an increased risk of AV block4.
Case continued
The patient was admitted and had no further asystole after receiving atropine and intermittent transcutaneous pacing. He was accepted for a dual-chamber pacemaker insertion and was discharged with the diagnosis of syncope with sinus arrest and vagal overtones.
Take Home Points
- Patients presenting to the ER with new-onset syncope require a thorough history and physical exam to rule out cardiogenic causes.
- Validated clinical decision-making tools can be helpful to supplement clinical judgement for assessing the risk of a future cardiac event, identifying the need for a cardiology consult and creating a disposition plan.
References
- Runser LA, Gauer RL, Houser A. Syncope: Evaluation and Differential Diagnosis. Am Fam Physician. 2017;95(5):303-312. https://www.aafp.org/pubs/afp/issues/2017/0301/p303.html#:~:text=A%20standardized%20approach%20to%20syncope,%2C%20physical%20examination%2C%20and%20electrocardiography
- UpToDate. www.uptodate.com. https://www.uptodate.com/contents/syncope-in-adults-clinical-manifestations-and-initial-diagnostic-evaluation
- Sandhu RK, Raj SR, et al. Canadian Cardiovascular Society Clinical Practice Update on the Assessment and Management of Syncope. Can J Cardiol. 2020;36(8):1167-1177. doi:10.1016/j.cjca.2019.12.023 https://www.onlinecjc.ca/article/S0828-282X(19)31549-1/fulltext
- Brignole M, Moya A, de Lange FJ, et al. 2018 ESC Guidelines for the diagnosis and management of syncope. Eur Heart J. 2018;39(21):1883-1948. doi:10.1093/eurheartj/ehy037https://academic.oup.com/eurheartj/article/39/21/1883/4939241?login=false
- Dakkak W, Doukky R. Sick Sinus Syndrome. In: StatPearls. Treasure Island (FL): StatPearls Publishing; July 18, 2022. https://www.ncbi.nlm.nih.gov/books/NBK470599/
Under pressure: Anorectal abscesses… to drain or not to drain?
Resident Clinical Pearl
Victoria Landry
iFMEM R3
Reviewed by Dr. J Mekwan
Copy Edited by Dr. J Vonkeman
PDF Download: EMSJ Anorectal Abscess by VLandry
Case
A 57yo male presents to the emergency department with complaints of a lump near his rectum and pain with sitting which developed over the past week. He is afebrile with normal vital signs. He tells you that about 6 months ago this same thing occurred and drainage was attempted in the ED but was unsuccessful. A colorectal surgeon subsequently drained it successfully under local anesthetic in clinic later the same day. He has had no recurrence of symptoms until the past week. He smokes and has hypertension controlled with medication but is otherwise healthy.
He denies pain with defecation and has not had any rectal bleeding nor changes in bowel habits. He feels otherwise well and denies fevers or chills.
On exam you find a tender firm mass in the subcutaneous tissue lateral to his rectum on the left side. There is minimal overlying erythema and no fluctuance.
Key Point #1: Always do a Digital Rectal Exam
- Palpate in all directions to localize area of tenderness1
- Should be unremarkable after you get past the anal verge2 – if tenderness, mass, induration past anal verge, do a CT scan to assess for deeper abscess
You think back to your perirectal anatomy and recall the spaces where abscesses can develop.
Figure 1: Transverse anorectal anatomy3
Figure 2: Longitudinal anorectal anatomy3
Perianal | Ischiorectal | Intersphincteric | Supralevator | Postanal | |
Incidence | 40-45% | 20-25% | 20-25% | <5% | 5-10% |
Location | Outside anal verge, red, swollen, fluctuant, easily palpable at anal verge | Between rectum and ischial tuberosity, outside sphincters, palpable through rectal wall or lateral to anal verge on buttocks | Lower rectum, between sphincters, inferior to levator ani (tender indurated mass in rectum) | Above levator ani (tender indurated mass in rectum) | Posterior to rectum, Deep to external sphincter, inferior to levator ani |
Symptoms | Painful perianal mass | Buttock pain | Rectal fullness, throbbing, worse with defecation | Perianal and buttock pain | Rectal fullness and pain near coccyx |
Fever, ↑WBC | No | Possibly | Possibly | Yes | Yes |
Fistula formation | ++ | + | +++ | +++ | – |
ED I&D | Yes | Possibly: I&D/needle aspiration only if abscess is superficial and fluctuant | No | No:
Consult surgery for urgent drainage |
No |
Table 1: Types of abscesses3
** caution as mass may be bigger/deeper than anticipated – prudent to defer to surgery for their expertise
Figure 3: Anorectal abscess locations4
Key Point #2: Get a CT scan to define the abscess for any of the following2
- Unable to see the abscess superficially
- Patient is unable to tolerate the DRE due to significant pain
- Induration, bogginess or tenderness in the supralevator space (above the sphincter muscle)
- If the extent of the abscess is uncertain4
Note: can use POCUS to evaluate location of abscess, but caution against false reassurance as to extent/depth, and safer to rely on palpable fluctuant mass to determine if I&D is safe
Figure 4: Perianal abscess on CT1
Management5
- Simple, isolated, fluctuant perianal abscess4
- Bedside I&D
- Goal is to relieve the pus under pressure2
- Ischiorectal abscess2
- Can consider I&D only if superficial, but prudent to get a CT first
- Consult surgery for their expertise
- Intersphincteric, Supralevator, Postanal
- CT to define the abscess
- Consult surgery
Key Point #3: Err on the side of caution
Only do I&D in the ED if the following criteria are met3 [3]
- Perianal abscess (+/- ischiorectal) is small and superficial
- Patient
- Is Well-appearing
- Is Cooperative
- Has no complicating factors (DM, immune compromise etc.)
Incision and Drainage of simple perianal abscess2
- Local anesthetic – lidocaine with epinephrine
- Infiltrate superficial skin where you will poke with needle
- occasionally procedural sedation is needed3
- Needle poke +/- aspiration (18guage) or pinpoint incision over painful region to localize purulent pocket4
- Inject more local anesthetic2
- Enlarge the incision
- Make incision as close to anal verge as possible to minimize the length of any potential fistula2,5,6
- Cruciate (with trimming of the flaps) or elliptical incision over fluctuant part of abscess is preferred over a linear incision to keep incision open and draining without painful packing2
- If linear only, will need packing to prevent premature closure
- Note: loop drainage technique not recommended for I&D in the ED7
- Break up loculations with finger (increased tactile feedback and better control) or hemostat +/- irrigation with saline7
- Cover with bulky dressing4
- Ideally, close follow up until complete healing (up to 8wks) to monitor for recurrence and for fistula formation5
- Uncomplicated perianal abscesses do not require antibiotics after successful drainage2.
Figure 5: Cruciate incision4
Instruct the patient to WASH8
- W – warm water sitz baths 5-10min BID-QID PRN, with Epsom salts (start the day after I&D)
- Water >40°C helps decrease anal canal pressure
- A – analgesics (NSAIDs, topical 1-2% lidocaine gel)
- S – stool softeners (PEG, senna)
- H – high fiber diet +/- fiber supplement
- Uncomplicated perianal abscesses do not require antibiotics after successful drainage2.
Indications for antibiotics (+/- tetanus +/- admission to hospital with surgical consult)4
- Surrounding cellulitis
- Immune compromise
- Valvular heart disease
- Diabetes
- Systemic symptoms (Fever, ill appearing, leukocytosis)
- Elderly
Note: Send off a wound culture before giving antibiotics
Antibiotic choice5:
- Systemic: piperacillin-tazobactam
- Oral: Amoxicillin-clavulanate or Metronidazole + ciprofloxacin
A word on fistulas
- Fistulas are a connection between two epithelium-lined surfaces, characterized by persistent or recurrent anal drainage. They are seen in Crohn’s, TB, cancer, FB reactions, and as a complication of anorectal abscesses. Treatment is surgical3
- ~50% of anorectal abscesses form a fistula overtime2
- Suggest surgical consultation after drainage of perianal abscess as fistula formation is common4
- Fistulas may be missed on CT scan; MRI is more sensitive for diagnosis2
Take home points:
- Always do a rectal exam as part of the initial evaluation
- Have a low threshold to get a CT scan to define the abscess
- Reserve I&D in the ED for perianal abscesses that are visible, superficial and fluctuant
References
- Farah, Jennifer, Mason, Jessica, and Werner, Jessie, “Perirectal Abscess & Pilonidal Cyst.” [Online]. Available: https://www.emrap.org/episode/gastro/perirectal
- Jhun, Paul and Cologne, Kyle, “Anorectal Infections,” HIPPO EMRAP, vol. 15, no. 9, pp. 17–18, Sep. 2015.
- Parrillo, “Anorectal Emergencies,” presented at the EMRAP, Temple University Hospital EM Residency, Feb. 2004. [Online]. Available: https://www.emrap.org/episode/september2004/anorectal
- Berberian J.G., & Burgess B.E. Tintinalli J.E., & Ma O, & Yealy D.M., & Meckler G.D., & Stapczynski J, & Cline D.M., & Thomas S.H.(Eds.), “Anorectal disorders,” in Tintinalli’s Emergency Medicine: A Comprehensive Study Guide, 9e, McGraw Hill, 2020. [Online]. Available: https://accessmedicine-mhmedical-com.ezproxy.library.dal.ca/content.aspx?bookid=2353§ionid=219642697
- Streitz Matthew, Long Brit, “Anorectal Disease,” in CorePendium, Burbank, CA: CorePendium, LLC, 2022. [Online]. Available: https://www.emrap.org/corependium/chapter/reclLjrt5HvPGSIDv/Anorectal-Disease#h.d78nqbylr3x
- Bleday, Ronald, Perianal and perirectal abscess. uptodate.com, 2022. [Online]. Available: https://www.uptodate.com/contents/perianal-and-perirectal-abscess
- Cavanaugh, Megan and Ormon, Rob, “Anorectal Disorders.” [Online]. Available: https://www.emrap.org/episode/april2011/anorectal
- Lipp, Chris, “Anorectal Disorders.” [Online]. Available: https://canadiem.org/crackcast-e096-anorectal-disorders/
Unvexing the VExUS Score – An Overview
Unvexing the VExUS Score – An Overview
PoCUS Clinical Pearl
Dr Steven Chen
DalEM PoCUS Elective
PGY2 Internal Medicine, University of Toronto
Reviewed: Dr David Lewis
Copyedited: Dr David Lewis
Introduction:
The pursuit of a rapid and objective measure of volume status has always been a vexing problem for clinicians as proper fluid management is pivotal for patient outcomes. In recent years, there has been increased attention towards the concept of “fluid-responsive” as liberal fluid boluses can often be associated with poor outcomes as a result of systemic congestion. 1
In the POCUS community, while Inferior Vena Cava (IVC) measurements have promise in assessing central venous pressure, the subsequent translation towards “volume responsiveness” has been met with many other limitations. For one, it did not account for venous congestion at other organ levels such as the pulmonary, renal, or hepatic systems. 2,3
Venous excess ultrasound (VExUS) is a growing bedside ultrasound-based approach that aims to provide a more comprehensive assessment of venous congestion. This was initially described by Beaubien-Souligny et al. (2020) from a post-hoc analysis correlating ultrasound grading parameters with risk in development of AKI in cardiac surgery patients.4 The protocol serves to assess multiple sites of venous congestion, including the IVC, hepatic veins, portal veins and intrarenal veins. By assessing congestion in these multiple sites, the VExUS score has gained attraction in providing a more comprehensive assessment of systemic congestion. 4,5
View Acquisition:
The VExUS protocol is composed of four main components outlined below:
- IVC diameter
- Hepatic Vein Doppler Assessment
- Hepatic Portal Vein Doppler Assessment
- Intrarenal Vein Doppler Assessment
This can be performed using either the curvilinear probe (preferred) or the phased array probe. The patient should be positioned flat and supine on the bed to acquire the views. The table below depicts some suggested views where larger regions of the veins may be accessible for pulse wave doppler gating in reference to standardized sonography protocols. 6,7
Note: Reviewing the basics of pulse wave doppler will be needed prior to completing VExUS scans (not covered in this article).
Interpretation:
Interpretation of the VExUS grading system is well summarized in diagram below (sourced from POCUS1018) and takes some practice to differentiate normal from abnormal waveforms. Pulse wave doppler assessment is pursued only if the inferior vena cava is found plethoric, defined as greater or equal to 2cm. 4,5
Each of the hepatic, portal and renal veins are subsequently examined and classified as normal, mildly congested, or severely congested. The VExUS system has four grades: Grade 0 represents no congestion in any organ, Grade 1 represents only mild congestive findings, Grade 2 represents severe congestive findings in only one organ, and Grade 3 represents severe congestive findings in at least two out of three organ systems. 4,5
Source: POCUS1018
Some sample waveforms are shown below with comments to help with distinguishing normal from abnormal waveforms.
Evidence:
VExUS has also been shown to be reliable and reproducible, with good interobserver agreement in trained individuals and correlation with other measures of volume status such as central venous pressure.4,5 As the technique is growing in the POCUS literature, below is a table summarizing several recent studies exploring its application across numerous settings.
Study | Purpose | Results |
Beaubien-Souligny W, et al. (2020)4
Post-hoc analysis of a single centre prospective study in 145 patients
|
Initial model of VExUS grading system looking at association in development of AKI in cardiac surgery population | Association with subsequent AKI:
HR: 3.69 CI 1.65–8.24 p = 0.001; +LR: 6.37 CI 2.19–18.50 when detected at ICU admission, which outperformed central venous pressure measurements
|
Bhardwaj V, et al. (2020)9
Prospective cohort study of 30 patients in ICU setting
|
Prospective study on application of VExUS scoring on staging of AKI in patients with cardiorenal syndrome | Resolution of AKI injury significantly correlated with improvement in VExUS grade (p 0.003).
There was significant association between changes in VExUS grade and fluid balance (p value 0.006). |
Varudo R, et al. (2022)10
Case report of ICU patient with hyponatremia |
Application of VExUS in case report as rapid tool to help with volume status assessment in patient with complex hyponatremia | Overall VExUS grade 2, prompting strategy for diuresis with improvement |
Rolston D, et al. (2022)11
Observational study of 150 septic patients in single centre |
VExUS score performed on ED septic patients prior to receiving fluids with chart review done to determine if there is association with poorer outcomes | Composite outcome (mortality, ICU admission or rapid response activation):
VExUS score of 0: 31.6% of patients VExUS score of 1: 47.6% of patients VExUS score >1: 67.7% of patients (p: 0.0015) |
Guinot, PG, et al. (2022)12
Prospective observational study of 81 ICU patients started on loop diuretic therapy |
Evaluation of multiple scores to predict appropriate diuretic-induced fluid depletion (portal pulsatility index, renal venous impedance index, VExUS) | Baseline portal pulsatility index and renal venous impedance index were found to be superior predictors compared to VExUS.
The baseline VExUS score (AUC of 0.66 CI95% 0.53–0.79, p = 0.012) was poorly predictive of appropriate response to diuretic-induced fluid depletion. |
Menéndez‐Suso JJ, et al. (2023)13
Cross-sectional pilot study of 33 children in pediatric ICU setting |
Association of VExUS score with CVP in pediatric ICU | VExUS score severity was strongly associated with CVP (p<0.001) in critically ill children. |
Longino A, et al. (2023)14
Prospective validation study in 56 critically ill patients |
Validation looking at association of VExUS grade with right atrial pressure. | VExUS had a favorable AUC for prediction of a RAP ≥ 12 mmHg (0.99, 95% CI 0.96-1) compared to IVC
diameter (0.79, 95% CI 0.65–0.92). |
Pitfalls:
It should be kept in mind that numerous factors may affect interpretation of VExUS gradings.
For the IVC component, increased intra-abdominal pressure can affect measurements independently of the pressure in the right atrium or may be affected by chronic pulmonary hypertension. The hepatic vein may not show significant changes even in severe tricuspid regurgitation if the right atrium can still expand and contract normally. In thin healthy people and those with arteriovenous malformations, the portal vein can have a pulsatile flow without venous congestion. It is also important to note that for patients with underlying disease renal or liver parenchymal disease, venous doppler recordings may be less reliable. 3-5
Outside of physiologic factors, another limitation is the need for adequate training and familiarity in performing and interpreting the technique. While VExUS is fairly well protocolized, it requires proficiency with pulse wave doppler to perform accurately. As with any new technique, there is a risk of variability in technique and interpretation. To avoid misinterpretation, it is important to consider repeat tracings to ensure consistency of results and to consider findings within the overall clinical context of the patient.
Bottom line:
VExUS is a non-invasive ultrasound method for assessing venous congestion across multiple organ systems. While there are several physiologic limitations and results need to be used in adjunct with the clinical picture, studies have shown promise for VExUS to be incorporated as part of a physician’s toolkit to help with clinical decision making. 3-5
References
- Atkinson P, Bowra J, Milne J, Lewis D, Lambert M, Jarman B, Noble VE, Lamprecht H, Harris T, Connolly J, Kessler R. International Federation for Emergency Medicine Consensus Statement: Sonography in hypotension and cardiac arrest (SHoC): An international consensus on the use of point of care ultrasound for undifferentiated hypotension and during cardiac arrest. Canadian Journal of Emergency Medicine. 2017 Nov;19(6):459-70.
- Corl KA, George NR, Romanoff J, Levinson AT, Chheng DB, Merchant RC, Levy MM, Napoli AM. Inferior vena cava collapsibility detects fluid responsiveness among spontaneously breathing critically-ill patients. Journal of critical care. 2017 Oct 1;41:130-7.
- Koratala A, Reisinger N. Venous excess doppler ultrasound for the nephrologist: Pearls and pitfalls. Kidney Medicine. 2022 May 19:100482.
- Beaubien-Souligny W, Rola P, Haycock K, Bouchard J, Lamarche Y, Spiegel R, Denault AY. Quantifying systemic congestion with point-of-care ultrasound: development of the venous excess ultrasound grading system. The Ultrasound Journal. 2020 Dec;12:1-2.
- Rola P, Miralles-Aguiar F, Argaiz E, Beaubien-Souligny W, Haycock K, Karimov T, Dinh VA, Spiegel R. Clinical applications of the venous excess ultrasound (VExUS) score: conceptual review and case series. The Ultrasound Journal. 2021 Dec;13(1):1-0.
- Mattoon JS, Berry CR, Nyland TG. Abdominal ultrasound scanning techniques. Small Animal Diagnostic Ultrasound-E-Book. 2014 Dec 2;94(6):93-112.
- Standardized method of abdominal ultrasound [Internet]. Japanese society of sonographers. [cited 2023Apr12]. Available from: https://www.jss.org/english/standard/abdominal.html#Longitudinal%20scanning_2
- Dinh V. POCUS101 Vexus ultrasound score–fluid overload and venous congestion assessment.
- Bhardwaj V, Vikneswaran G, Rola P, Raju S, Bhat RS, Jayakumar A, Alva A. Combination of inferior vena cava diameter, hepatic venous flow, and portal vein pulsatility index: venous excess ultrasound score (VExUS score) in predicting acute kidney injury in patients with cardiorenal syndrome: a prospective cohort study. Indian journal of critical care medicine: peer-reviewed, official publication of Indian Society of Critical Care Medicine. 2020 Sep;24(9):783.
- Varudo R, Pimenta I, Blanco JB, Gonzalez FA. Use of Venous Excess UltraSound (VExUS) score in hyponatraemia management in critically ill patient. BMJ Case Reports CP. 2022 Feb 1;15(2):e246995.
- Rolston D, Li T, Huang H, Johnson A, van Loveren K, Kearney E, Pettit D, Haverty J, Nelson M, Cohen A. 204 A Higher Initial VExUS Score Is Associated With Inferior Outcomes in Septic Emergency Department Patients. Annals of Emergency Medicine. 2021 Oct 1;78(4):S82.
- Guinot PG, Bahr PA, Andrei S, Popescu BA, Caruso V, Mertes PM, Berthoud V, Nguyen M, Bouhemad B. Doppler study of portal vein and renal venous velocity predict the appropriate fluid response to diuretic in ICU: a prospective observational echocardiographic evaluation. Critical Care. 2022 Dec;26(1):1-1.
- Menéndez‐Suso JJ, Rodríguez‐Álvarez D, Sánchez‐Martín M. Feasibility and Utility of the Venous Excess Ultrasound Score to Detect and Grade Central Venous Pressure Elevation in Critically Ill Children. Journal of Ultrasound in Medicine. 2023 Jan;42(1):211-20.
- Longino A, Martin K, Leyba K, Siegel G, Gill E, Douglas I, Burke J. Prospective Validation of the Venous Excess Ultrasound “(VExUS)” Score.
Downloadable Format
Why are ER wait times so bad in Canadian cities?
In this new Globe and Mail Podcast, we hear from Dr. Paul Atkinson and others who continue to provide their insights into the issues relating to increased Emergency Department wait times and expand on the widely read article “Saving Emergency Medicine: Is less more?”
You can listen to this podcast here:
Further reading and podcasts relating to Saving Emergency Medicine can be accessed here:
EM Grand Rounds: Practical Points in Aboriginal Health
Dr. Tiffany O’Donnell (MD, CCFP, C. ISAM)
Andrea Paul-McCoy (Dalhousie NP Student, RN BN)
Resources:
Commonly Used Indigenous Terms & Phrases You May Not Be Familiar With
Approach to Inguinal and Femoral Hernias in the Emergency Department
Medical Student Pearl
Julia Short
Med 2
DMNB Class of 2025
Reviewed by Dr D Lewis
Copy Edited by Dr. J Vonkeman
PDF Download: EMSJ Approach to Inguinal and Femoral Hernias in the ED by JShort
Case
A 52-year-old male patient presents in the ER with a lump in their right groin. The lump protrudes when they cough and when laying on their left side, although it re-enters the abdomen on its own. You wonder if it could be a femoral or an inguinal hernia, and how to go about differentiating between the two.
Introduction
A hernia is defined as an organ, or part of an organ, that protrudes through the body wall in which it is normally contained. The etiology of a hernia can be due to congenital anatomical malformations or from acquired weakening of the body wall tissues. There are various subtypes of abdominal hernias, while groin hernias consist of inguinal and femoral hernias. Throughout their lifetime, males have a 27 to 43% chance of developing a groin hernia, while females have a 3 to 6% lifetime prevalence1. Although it is much more likely that a groin hernia is inguinal in nature (they account for 96% of groin hernias), it is clinically useful to identify and distinguish between the types of groin hernias. Additionally, there are important clinical features that must not be overlooked when characterizing a groin hernia.
Distinguishing inguinal from femoral hernias
An important landmark in determining the hernia origin is the inguinal ligament. Inguinal hernias protrude superior to the inguinal ligament, while femoral hernias present inferior to the inguinal ligament (Figure 1). This is because femoral hernias protrude from the femoral ring, located medial to the femoral vein. As a result, in males, femoral hernias will never course into the scrotum. Femoral hernias also present more lateral than inguinal hernias and may be difficult to differentiate from lymph nodes. Although they account for only 3% of all groin hernias, 40% of femoral hernias present as urgent due to bowel strangulation or incarceration1. Females are more likely to develop femoral hernias, while males are more likely to develop inguinal hernias.
Figure 1. Groin anatomy © 2023 UpToDate7
Distinguishing between direct and indirect inguinal hernias
Direct inguinal hernias originate medially, near the pubic tubercle and external inguinal ring. They protrude through Hesselbach’s triangle as a result of weakness in the floor of the inguinal canal. On exam, a bulge near the external (superficial) inguinal ring is suggestive of a direct inguinal hernia. In contrast, indirect inguinal hernias protrude near the midpoint of the inguinal ligament, at the internal (deep) inguinal ring (Figure 2). In males and females respectively, the internal inguinal ring is where the spermatic cord and round ligament exit the abdomen. A bulge in this area therefore suggests an indirect inguinal hernia. This type of hernia is the most common in all ages and sexes, accounting for approximately two thirds of all inguinal hernias2. In males, the indirect hernia often courses into the scrotum, which can be palpated if the patient strains or coughs. In contrast, it is rare for a direct hernia to course into the scrotum.
Figure 2. Anatomical comparison of direct and indirect inguinal hernias © 2020 Dr. Vaibhav Kapoor8
Clinical Approach
General considerations for investigating groin hernias include assessing the symptoms at presentation as well as any “red flag” physical findings. Patients commonly complain of dull or heavy types of discomfort when straining, which resolves when straining stops. Most groin hernias occur on the right side. Common physical findings include a bulge in the groin, which can indicate the type of hernia based on location relative to the inguinal ligament (Figure 3). However, in female or obese patients, the layers of abdominal wall may make the hernia more difficult to locate. In these cases, ultrasound or other imaging is needed to detect hernias. Clinicians should also determine if the hernia is reducible, or if the herniated bowel can be returned to the abdominal cavity when moderate pressure is applied externally.
Figure 3. Locations of femoral and inguinal hernias on examination © 2023 UpToDate7
Physical examination has a 76 to 92% sensitivity and 96% specificity for diagnosing groin hernias, although imaging may also be required1,2. Nausea, vomiting, fever, moderate-to-severe abdominal pain, localized tenderness, or bloating may indicate more sinister pathology such as bowel incarceration (when the hernia contents cannot return to the abdominal cavity), strangulation (when the blood supply to the involved bowel section is compromised) or necrosis.
Figure 5. CT images of A) femoral hernia (courtesy of Chris O’Donnell9 and B) inguinal hernia (courtesy of Erik Ranschaert10)
Management
Uncomplicated or asymptomatic hernias in males can be monitored through watchful waiting. Surgical repair is a definitive treatment for inguinal hernias and should be considered for symptomatic or complex hernias. If repair is needed for an uncomplicated inguinal hernia, a laparoscopic repair is recommended. Watchful waiting is not recommended for femoral hernias – these patients should have a laparoscopic repair (when anatomically feasible).
Manual reduction of the hernia can be performed by following the GPS Taxis technique. Taxis is a non-invasive technique for manual reduction of incarcerated tissues in a hernia to the original compartment5. “GPS” is an acronym to remind clinicians to be gentle, be prepared, and be safe when performing taxis5. Conscious sedation with intravenous diazepam and morphine is recommended for the procedure. Consider having an anesthetist present for the procedure if the patient is frail. Provide appropriate early resuscitation by monitoring vital signs, administering oxygen therapy and establishing IV access. Place the patient in Trendelenburg position. Begin the GPS Taxis technique by palpating the fascial defect around the base of the hernia and gently manipulating hernia contents back into the abdominal cavity. Use gentle manipulation pressure over 5-10 minutes until a gurgling sound is heard (indicating successful reduction of bowel).
Taxis guided by ultrasound may increase success rates for reduction.
https://sjrhem.ca/taxis-reduction-of-inguinal-hernia/
Figure 4. Colourized clip demonstrating PoCUS assisted Taxis reduction of an inguinal hernia11
It should be noted that the major contraindication to performing GPS Taxis is bowel strangulation within the hernia. A rare but serious complication of manual reduction is reduction en masse, when a loop of bowel remains incarcerated at the neck of the hernia after manual reduction6. This can lead to early strangulation, intestinal necrosis, sepsis, organ failure and death. Femoral hernias and indirect inguinal hernias are at higher risk of reduction en masse from manual reduction attempts.
References:
- UpToDate – Classification, clinical features, and diagnosis of inguinal and femoral hernias in adults
- Hammoud M, Gerken J. Inguinal hernia. StatPearls. 2022 Aug 15.
- UpToDate – Overview of treatment for inguinal and femoral hernia in adults
- Bates’ Guide to Physical Examination and History Taking, 12th ed. (pdf). Chapter 13: Male Genitalia and Hernias
- Pawlak M, East B, de Beaux AC. Algorithm for management of an incarcerated inguinal hernia in the emergency settings with manual reduction. Taxis, the technique and its safety. Hernia, 25, 1253-1258. 2021 May 25.
- Yatawatta A. Reduction en masse of inguinal hernia: a review of a rare and potentially fatal complication following reduction of inguinal hernia. BMJ Case Rep. 2017 Aug 7.
- UpToDate – Classification, clinical features, and diagnosis of inguinal and femoral hernias in adults
- Kapoor, V. Difference between and inguinal and umbilical hernia. 2020. Retrieved from: https://www.drvaibhavkapoor.com/difference-between-inguinal-and-umbilical-hernia.html
- Patel, MS. Femoral hernia. Radiopaedia. 2022 Dec 28. Retrieved from: https://radiopaedia.org/articles/femoral-hernia
- Fahrenhorst-Jones, T. Inguinal hernia. Radiopaedia. 2022 Apr 12. Retrieved from: https://radiopaedia.org/articles/inguinal-hernia
- PoCUS assisted Taxis reduction of an inguinal hernia. Video obtained courtesy of Dr. David Lewis.
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